The incidence of Metabolic Syndrome (MetS) has increased dramatically over the past decade, and research has shown that some prevalent forms of MetS have at least one genetic component. Multiple gene targets must be involved in the pathogenesis and progression of this disease, and their identification would enable evaluation for genetic susceptibility to MetS.
Epigenetic alterations in utero are also affected by environmental factors that cause phenotypic changes later in life. Within the human p…